It can be fascinating to notice that when the DCM-leading to mutation K36Q in cTnI was launched in the presence of Ca2+, the simulation showed that Ser 22 and 23 not interacted closely with cTnC, in accord with our hypothesis the Ca2+-cTnC-cTnI N terminus conversation is unique and it is destabilized right by phosphorylation as well as allosterical

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